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Aging Overview

External factors

External factors: Costunolide
Aging type: Accelerate
Aging characteristic:
Category: Chemical compounds
Phenotype: Glioblastoma
Experimental category: HL
Tissue type: --
Cell name: A172,U87 MG
PMID: 27148686
Experiment: SA-β-gal activity assay
Description: Costunolide-induced increase in β-gal-positive cells, indicative of senescence, was diminished in the presence of ROS inhibitor.



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Regulatory relationship

Target gene: GS(P)//TKT
R-EF-Target gene: Downregulation//Downregulation
Official symbol(s): APC//TKT
Target gene experiment: Western blot
Target gene description: Costunolide-mediated decrease in tumor volume was accompanied by decreased expression of TERT, Nrf2, G6PD, TKT, and GS(P) levels . Costunolide-induced senescence was accompanied by decreased expression of GS(P) and increased glycogen accumulation in an ROS-dependent manner. The extent of glycogen accumulation was cell line dependent.


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Regulatory pathway: --
R-EF-Pathway: --
Pathway experiment: --
Pathway description: --


Aging network

Annotation:

The green line represents Upregulation.

The purple line represents Downregulation.

The orange line represents Activation.

The yellow line represents Inhibition.

The gray line represents Unclear.




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