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Aging Overview

External factors

External factors: Methoxyamine
Aging type: Accelerate
Aging characteristic:
Category: Chemical compounds
Phenotype: Colorectal cancer
Experimental category: L
Tissue type: --
Cell name: RKO
PMID: 16648559
Experiment: SA-β-gal activity assay//Cell morphological analysis
Description: Examined senescence-activated β-gal activity following these treatments. Senescence-activated β-gal activity was examined on days 5 to 8 following ionizing radiation and was found to be positive in IUdR- and IUdR/methoxyamine–pretreated cells. Spot senescence-activated β-gal positive cells were also found in the control and methoxyamine-pretreated cells;During the course of the experiments, we noted that after ionizing radiation, IUdR- and IUdR/methoxyamine–pretreated cells exhibited senescence-like morphologic features .



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Regulatory relationship

Target gene: P53//P21//RB
R-EF-Target gene: Upregulation//Upregulation//Activation
Official symbol(s): P53//P21//RB
Target gene experiment: Western blot
Target gene description: Indeed, both accumulation of p53 and induction of p21, the major mediator of p53-dependent senescence, increase more significantly in IUdR/methoxyamine–pretreated cells at both early and later times following ionizing radiation. Furthermore, IUdR/methoxyamine–pretreated cells show reduced hyperphosphorylated Rb(S780) (inActivate form) but greater hypophosphorylated Rb (Activate form) compared with IUdR-pretreated cells .


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Regulatory pathway: --
R-EF-Pathway: --
Pathway experiment: --
Pathway description: --


Aging network

Annotation:

The green line represents Upregulation.

The purple line represents Downregulation.

The orange line represents Activation.

The yellow line represents Inhibition.

The gray line represents Unclear.




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